HOME   |    PDF   |   


Title

Linking gut microbiota with human diseases

Authors

Udaya Kumar Vandana1, Naseema Hayat Barlaskar1, Abu Barkat Md. Gulzar1, Islamul Hoque Laskar1, Diwakar Kumar2, Prosenjit Paul1, Piyush Pandey2, P.B. Mazumder1*

 

Affiliation

1Department of Biotechnology, Assam University, Silchar, Assam, India; 2Department of Microbiology, Assam University, Silchar, Assam, India;

 

Email

 Pranab Behari Mazumder - Email: pbmmmbl@gmail.com; *Corresponding author;

Udaya Kumar Vandana - uday21microbe@gmail.com, Naseema Hayat Barlaskar - naseema788@gmail.com; Abu Barkat Mohammed Gulzar - gulzar.hussain282@gmail.com, Islamul Hoque Laskar - islamul9393@gmail.com, Diwakar Kumar - diwakar11@gmail.com,
Prosenjit Paul - prosenjit.paul77@gmail.com, Piyush Pandey - piyushddn@gmail.com, P.B. Mazumder - pbmmmbl@gmail.com

 

Article Type

Research Article

 

Date

Received January 2, 2020; Accepted February 20, 2020; Published February 29, 2020

 

Abstract

The human gut is rich in microbes. Therefore, it is of interest to document data to link known human diseases with the gut microbiota. Various factors like hormones, metabolites and dietary habitats are responsible for shaping the microbiota of the gut. Imbalance in the gut
microbiota is responsible for the pathogenesis of various disease types including rheumatoid arthritis, different types of cancer, diabetes mellitus, obesity, and cardiovascular disease. We report a review of known data for the correction of dysbiosis (imbalance in microbe
population) towards improved human health.

 

Keywords

Bacteria; dysbiosis; immunoglobulin; Gut microbiome; short chain fatty acids; cancer

 

Citation

Vandana et al. 16(2): 196-208 (2020) 

 

Edited by

P Kangueane

 

ISSN

0973-2063

 

Publisher

Biomedical Informatics

 

License

This is an Open Access article which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. This is distributed under the terms of the Creative Commons Attribution License.